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Monday, December 5, 2016

Named the main danger posed by alcohol


The body of a person suffering from alcoholism undergoing change, not only even more exacerbated his alcohol dependence, but in General, deprives it of the ability to fully control their behavior. The fact that people who abuse alcohol, decreases the production of an important enzyme known as PRDM2, explain the experts from Germany University.


photo: pixabay.com

The enzyme is produced of the same name PRDM2 gene, but its role is still can not be called fully studied. However, in the new study, scientists have discovered that PRDM2 plays an important role in ensuring the normal operation of the frontal lobes of the brain. How figured out the experts in the course of experiments on laboratory rats, alcohol inhibits the production of PRDM2, thus complicating the work of the frontal lobe. This, in turn, leads to the fact that the behavior of someone who suffers from alcoholism, change.

The frontal lobe of the brain helps a person to control their immediate impulses. It allows you to think several steps ahead before you do something. For example, a person who is given the opportunity to drink a cold beer on a hot day, with a high probability think twice before you drink it if there will be important work. At the same time, if due to lack of PRDM2 his self-control will be weakened, man would not build a complex logical chain and drink beer immediately, just because he is hot. In the future, he will not even need to invent an excuse of this kind.

Experts say that alcoholism is a dangerous vicious circle — the more a person drinks, the less the body produces PRDM2, and this, in turn, leads to the fact that his desire to quit drinking even faster eroding. The lack of PRDM2 is aggravated not only alcohol dependence, but in General makes people more impulsive and less capable of self-control.

A new study published in the journal Molecular Psychiatry, published by Nature publishing company, one of the largest in the world in the production of scientific publications.

source

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